1,4-Dicaffeoylquinic acid
Selleck Chemicals
SKU:E7286
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1,4-Dicaffeoylquinic acid is a modulator linked to NF-kB Signaling and Immune Signaling and related pathway-oriented research. In practice, this places the compound in experiments that measure inflammatory transcriptional control and stress-responsive survival signaling and cytokine-mediated signaling and inflammatory effector responses in immunology and inflammation models.
The pathway annotation connects 1,4-Dicaffeoylquinic acid to NF-kB Signaling and Immune Signaling, supporting experiments that monitor inflammatory transcriptional control and stress-responsive survival signaling and cytokine-mediated signaling and inflammatory effector responses across biochemical, cellular, or phenotypic assay formats. This context is compatible with NF-kB reporter, cytokine, and inflammation assays and immune activation, cytokine-response, and mediator-profiling assays, as well as transcriptional, biochemical, or phenotypic comparisons linked to the annotated pathway state. In immunology and inflammation models, these readouts can be combined with viability, reporter, localization, biochemical conversion, or morphology endpoints to refine experimental interpretation.
Research Applications
- Pathway perturbation studies connected to NF-kB Signaling and Immune Signaling
- Context-dependent response profiling and mechanism comparison
- Phenotypic profiling in immunology and inflammation models
- Combination studies with orthogonal perturbagens
Overall, 1,4-Dicaffeoylquinic acid is well suited to pathway-oriented studies that need a defined compound input for NF-kB Signaling and Immune Signaling readouts in immunology and inflammation models. This profile is suited to mechanistic follow-up, comparative profiling, and assay optimization under defined exposure conditions.
- Pathways:
- NF-kB Signaling • Immune Signaling
- Research Area:
- Immunology • Inflammation
- CAS No.:
- 1182-34-9
- Molecular Weight:
- 516.45
- Storage Temperature:
- -20°C
For Research Use Only. Not intended for diagnostic or therapeutic use.
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