Ginkgolic Acid (C13:0)

Ginkgolic Acid (C13:0) is an inhibitor of Histone Acetyltransferases used in studies of Protein Degradation / Proteasome and Epigenetic Regulation. It is especially relevant when investigators need a named chemical input and interpretable readouts connected to ubiquitin-dependent turnover, proteostasis, and targeted protein degradation and chromatin-state control, histone modification, and transcriptional regulation in cancer, epigenetics, and protein homeostasis models.

By inhibiting Histone Acetyltransferases, Ginkgolic Acid (C13:0) can be used to examine ubiquitin-dependent turnover, proteostasis, and targeted protein degradation and chromatin-state control, histone modification, and transcriptional regulation. The epigenetic regulator annotation adds relevance to chromatin, epigenetic, and gene-expression profiling, together with downstream-response mapping in the same experimental setting. In cancer, epigenetics, and protein homeostasis models, these readouts can be combined with viability, reporter, localization, biochemical conversion, or morphology endpoints to refine experimental interpretation.

Research Applications

• Target-focused assays involving Histone Acetyltransferases
• Pathway perturbation studies connected to Protein Degradation / Proteasome and Epigenetic Regulation
• Concentration-response inhibition and target-dependence studies
• Chromatin, epigenetic, and gene-expression profiling

Overall, Ginkgolic Acid (C13:0) is appropriate when a defined chemical perturbant is needed to connect Histone Acetyltransferases with measurable biochemical, transcriptional, electrophysiological, imaging, or phenotypic readouts in cancer, epigenetics, and protein homeostasis models. This profile is suited to mechanistic follow-up, comparative profiling, and assay optimization under defined exposure conditions.