Procaine HCl

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Selleck Chemicals

SKU:S4023-50MG

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Procaine HCl is an inhibitor of Sodium Channels and NMDAR used in studies of Calcium Signaling and Neurotransmission. In practice, this places the compound in experiments that measure calcium flux, excitability, and secretion-linked signaling and synaptic signaling, receptor activity, and neuronal excitability in cardiovascular and neuroscience models.

By inhibiting Sodium Channels and NMDAR, Procaine HCl can be used to examine calcium flux, excitability, and secretion-linked signaling and synaptic signaling, receptor activity, and neuronal excitability. The ion channel annotation adds relevance to electrophysiology, ion-flux, and membrane-potential studies, together with downstream-response mapping in the same experimental setting. In cardiovascular and neuroscience models, these readouts can be combined with viability, reporter, localization, biochemical conversion, or morphology endpoints to refine experimental interpretation.

Research Applications

  • Target-focused assays involving Sodium Channels and NMDAR
  • Pathway perturbation studies connected to Calcium Signaling and Neurotransmission
  • Concentration-response inhibition and target-dependence studies
  • Electrophysiology, ion-flux, and membrane-potential studies

Overall, Procaine HCl is appropriate when a defined chemical perturbant is needed to connect Sodium Channels and NMDAR with measurable biochemical, transcriptional, electrophysiological, imaging, or phenotypic readouts in cardiovascular and neuroscience models. This profile is suited to mechanistic follow-up, comparative profiling, and assay optimization under defined exposure conditions.

Targets:
Sodium Channels • NMDAR
Target Class:
Ion Channel
Pathways:
Calcium Signaling • Neurotransmission
Research Area:
Cardiovascular • Neuroscience
CAS No.:
51-05-8
Molecular Weight:
272.77
Formula:
C₁₃H₂₀N₂O₂·HCl
SMILES:
CCN(CC)CCOC(=O)C1=CC=C(C=C1)N.Cl
Storage Temperature:
-20°C

For Research Use Only. Not intended for diagnostic or therapeutic use.
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